By Atul Mehta, Victor Hoffbrand
Haematology at a look offers a concise and available creation to haematology. It follows the easy-to-use 'at a look' structure of double-page spreads comprising transparent, memorable diagrams illustrating the foremost proof and crucial textual details. It starts off with a proof of standard blood cells and regimen laboratory checks. It then reports quite a lot of blood disorders.This new version of Haematology at a Glance:Contains complete color all through provides the main evidence followed via transparent diagrams, photographs and pictures Highlights the latest advancements within the box, together with insurance of transplantation know-how contains new chapters on : The facet impacts of drug remedy being pregnant and neonatological elements of haematology sensible tactics Haematology at a look will entice clinical scholars, junior medical professionals on origin programmes, haematology technicians and nursing and different healthiness care scholars with an curiosity in haematology.Reviews of final edition“Haematology at a look is a invaluable ebook for basic studying in addition to for revision, and i'm certain that i'm going to check with it for years to come.”The Royal collage of Pathologists Bulletin“It quite is excellent, in case you don’t think me, decide it up and glance yourself,”Black Bag – Bristol scientific tuition Gazette
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Additional info for Haematology at a Glance, 2nd edition (At a Glance)
Epidemiological evidence implicates Epstein–Barr virus in Burkitt’s lymphoma and less strongly with Hodgkin’s disease. Helicobacter pylori infection of the stomach predisposes to gastric lymphoma. 3 Ionizing radiation causes DNA mutation and increases the risk of haematological neoplasia. g. benzene and organochemicals, may predispose to leukaemia and myelodysplasia (MDS). 5 Drugs. g. melphalan, mustine) and other forms of chemotherapy predispose to MDS or acute myeloid leukaemia. g. ABL translocation from chromosome 9 to the breakpoint cluster region (BCR) on chromosome 22 to form BCR-ABL in CML, t(9;22)) (see Fig.
G. carcinoma Infiltration by abnormal tissue Myelofibrosis Rarely, Gaucher’s disease, amyloidosis, osteopetrosis Ineffective haemopoiesis Myelodysplasia, megaloblastic anaemia Laboratory findings • Anaemia, leucopenia and thrombocytopenia (pancytopenia) of varying severity. • Blood film typically shows no abnormal cells. g. circulating leukaemic blast cells. • Bone marrow aspirate and trephine biopsy are required to define cause (Fig. 19a). g. g. within an enlarged spleen). g. drugs. • Support care with appropriate blood components (see Chapter 41) and antimicrobials (see Chapter 43).
Pneumococcal infections may lead to pneumonia and meningitis. Infarction of intestinal mucosa predisposes to Salmonella infection and osteomyelitis may result. • Other clinical features include pigment gallstones with cholecystitis, chronic leg ulcers, avascular necrosis of the femoral and humeral heads (Fig. 18b) or other bones, cardiomyopathy, proliferative retinopathy and renal papillary necrosis (leading to polyuria, failure to concentrate urine and tendency to dehydration). Laboratory features • Haemoglobin level is 7–9 g/dL but symptoms of anaemia are usually mild (the O2 dissociation curve of Hb S is shifted to the right).